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ljenkinssDate: Saturday, 16.11.2013, 11:37 | Msg # 1
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Current evidence suggests that the pathogenesis of primary dysmenorrhea is due to prostaglandin F2alpha (PGF2alpha), a potent myometrial stimulant and vasoconstrictor, in the secretory endometrium.10 The response to prostaglandin inhibitors in patients with dysmenorrhea supports the assertion that dysmenorrhea is prostaglandin mediated. Substantial evidence attributes dysmenorrhea to prolonged uterine contractions and decreased blood flow to the myometrium. <a href=http://buymedsonline.org/products/buy-lioresal-online.htm>have a peek here</a>
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o Deep peritoneal disease is caused by infiltration of the uterosacral ligaments and rectovaginal septum by endometriotic nodules. Tethering of the uterus can lead to fixed retroversion. Dyspareunia is an important feature. <a href=http://buymedsonline.org/products/buy-famvir-online.htm>http://buymedsonline.org/products/buy-famvir-online.htm</a>
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* Chronic pain: The most important point to remember is that the degree of visible endometriosis has no correlation with the degree of pain or other symptomatic impairment.10 However, pain does correlate with the depth of tissue infiltration.11,12 Midline disease is generally believed to be more painful than lateral disease. <a href=http://buymedsonline.org/products/buy-apcalis-oral-jelly-online.htm>have a peek at this web-site</a>
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